McGrath LM, Braaten EB, Doty ND, et al. Extending the ’cross-disorder’ relevance of executive functions to dimensional neuropsychiatric traits in youth. J Child Psychol Psychiatry. 2016;57(4):462-71. doi:10.1111/jcpp.12463
Ruderfer DM, Fanous AH, Ripke S, et al. Polygenic dissection of diagnosis and clinical dimensions of bipolar disorder and schizophrenia. Mol Psychiatry. 2014;19(9):1017-24. doi:10.1038/mp.2013.138
Sumner JA, Duncan L, Ratanatharathorn A, Roberts AL, Koenen KC. PTSD has shared polygenic contributions with bipolar disorder and schizophrenia in women. Psychol Med. 2016;46(3):669-71. doi:10.1017/S0033291715002135
Ostacher MJ, Iosifescu DV, Hay A, Blumenthal SR, Sklar P, Perlis RH. Pilot investigation of isradipine in the treatment of bipolar depression motivated by genome-wide association. Bipolar Disord. 2014;16(2):199-203. doi:10.1111/bdi.12143
Song J, Bergen SE, Di Florio A, et al. Genome-wide association study identifies SESTD1 as a novel risk gene for lithium-responsive bipolar disorder. Mol Psychiatry. 2016;21(9):1290-7. doi:10.1038/mp.2015.165
Duncan LE, Holmans PA, Lee PH, et al. Pathway analyses implicate glial cells in schizophrenia. PLoS One. 2014;9(2):e89441. doi:10.1371/journal.pone.0089441
Bigdeli TB, Ripke S, Bacanu SA, et al. Genome-wide association study reveals greater polygenic loading for schizophrenia in cases with a family history of illness. Am J Med Genet B Neuropsychiatr Genet. 2016;171B(2):276-89. doi:10.1002/ajmg.b.32402
Gilman SE, Ni MY, Dunn EC, et al. Contributions of the social environment to first-onset and recurrent mania. Mol Psychiatry. 2015;20(3):329-36. doi:10.1038/mp.2014.36
Okbay A, Beauchamp JP, Fontana MA, et al. Genome-wide association study identifies 74 loci associated with educational attainment. Nature. 2016;533(7604):539-42. doi:10.1038/nature17671
Georgieva L, Rees E, Moran JL, et al. De novo CNVs in bipolar affective disorder and schizophrenia. Hum Mol Genet. 2014;23(24):6677-83. doi:10.1093/hmg/ddu379