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Stanley Center Therapeutics

Medicine. The future of psychiatric research: genomes and neural circuits.

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Akil H, Brenner S, Kandel E, et al. Medicine. The future of psychiatric research: genomes and neural circuits. Science. 2010;327(5973):1580-1. doi:10.1126/science.1188654.

Support of association between BRD1 and both schizophrenia and bipolar affective disorder.

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Nyegaard M, Severinsen JE, Als TD, et al. Support of association between BRD1 and both schizophrenia and bipolar affective disorder. Am J Med Genet B Neuropsychiatr Genet. 2010;153B(2):582-591. doi:10.1002/ajmg.b.31023.

Chemical genetics identifies small-molecule modulators of neuritogenesis involving neuregulin-1/ErbB4 signaling.

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Kuai L, Wang X, Madison JM, Schreiber SL, Scolnick EM, Haggarty SJ. Chemical genetics identifies small-molecule modulators of neuritogenesis involving neuregulin-1/ErbB4 signaling. ACS Chem Neurosci. 2010;1(4):325-342. doi:10.1021/cn900046a.

Tripping the HCN breaker.

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Lipscombe D, Pan JQ. Tripping the HCN breaker. Neuron. 2009;62(6):747-50. doi:10.1016/j.neuron.2009.06.003.

A genomewide association study of response to lithium for prevention of recurrence in bipolar disorder.

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Perlis RH, Smoller JW, Ferreira MAR, et al. A genomewide association study of response to lithium for prevention of recurrence in bipolar disorder. Am J Psychiatry. 2009;166(6):718-25. doi:10.1176/appi.ajp.2009.08111633.

Identifying relationships among genomic disease regions: predicting genes at pathogenic SNP associations and rare deletions.

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Raychaudhuri S, Plenge RM, Rossin EJ, et al. Identifying relationships among genomic disease regions: predicting genes at pathogenic SNP associations and rare deletions. PLoS Genet. 2009;5(6):e1000534. doi:10.1371/journal.pgen.1000534.

Collaborative genome-wide association analysis supports a role for ANK3 and CACNA1C in bipolar disorder.

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Ferreira MAR, O’Donovan MC, Meng YA, et al. Collaborative genome-wide association analysis supports a role for ANK3 and CACNA1C in bipolar disorder. Nat Genet. 2008;40(9):1056-8. doi:10.1038/ng.209.

Whole-genome association study of bipolar disorder.

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Sklar P, Smoller JW, Fan J, et al. Whole-genome association study of bipolar disorder. Mol Psychiatry. 2008;13(6):558-69. doi:10.1038/sj.mp.4002151.

Analysis of high-resolution HapMap of DTNBP1 (Dysbindin) suggests no consistency between reported common variant associations and schizophrenia.

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Mutsuddi M, Morris DW, Waggoner SG, Daly MJ, Scolnick EM, Sklar P. Analysis of high-resolution HapMap of DTNBP1 (Dysbindin) suggests no consistency between reported common variant associations and schizophrenia. Am J Hum Genet. 2006;79(5):903-9. doi:10.1086/508942.

Recurrence risks for schizophrenia in a Swedish national cohort.

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Lichtenstein P, Björk C, Hultman CM, Scolnick E, Sklar P, Sullivan PF. Recurrence risks for schizophrenia in a Swedish national cohort. Psychol Med. 2006;36(10):1417-25. doi:10.1017/S0033291706008385.